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test method blend components characteristic resid 1 resid 2 resid 3 resid 4 resid 5 decant oil vgo lco hco api gravity (Cell Signaling Technology Inc)

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Cell Signaling Technology Inc test method blend components characteristic resid 1 resid 2 resid 3 resid 4 resid 5 decant oil vgo lco hco api gravity
Test Method Blend Components Characteristic Resid 1 Resid 2 Resid 3 Resid 4 Resid 5 Decant Oil Vgo Lco Hco Api Gravity, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 93/100, based on 23 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/api+2/CHD4+Rabbit+mAb/us12421467-358-2-54
Average 93 stars, based on 23 article reviews

test method blend components characteristic resid 1 resid 2 resid 3 resid 4 resid 5 decant oil vgo lco hco api gravity - by Bioz Stars, 2026-10

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Article Title: Assessment of tantalum nanoparticle-induced MC3T3-E1 proliferation and underlying mechanisms
Article Snippet: Antibodies against LC3B, (p-)PI3K, (p-)Akt, (p-)mTOR, β-actin, and LY294002 (PI3K inhibitor, LY), Triciribine (Akt inhibitor, API) were all purchased from Cell Signaling Technology (CST, Beverly, MA, USA).

Article Title: Process for treating a hydrocarbon-containing feed
Article Snippet: %) 10.9 density (g/ml) (15.6° C.) 1.01 viscosity at 38° C. (cSt) 8357 API Gravity (15.6° C.) 8.5 TAN-E (ASTM D664) (mg KOH/g) 3.91 Boiling Range Distribution Initial boiling point-204° C. (400° F.) 0 (wt.

Residue:

Article Title: Low sulfur fuel oil blends for stability enhancement and associated methods
Article Snippet: .. TABLE VI Test Method Blend Components Characteristic Resid 1 Resid 2 Resid 3 Resid 4 Resid 5 Decant Oil VGO LCO HCO API Gravity @ ~60° F. 5.8 11.9 12.9 14.3 13.9 −0.3 25.3 39.0 39.0 Density @ ~15° C. (g/ml) 0.999 0.987 0.949 0.939 0.960 1.049 0.900 0.830 0.830 Viscocisty @ ~50° C. (cSt) 473.78 355.43 2234.82 888.93 10116.20 189.68 23.35 5.00 35.06 Sulfur (wt %) 1.74 2.51 0.42 1.38 1.59 0.30 0.05 0.05 0.17 Flash Point (° C.) 178.0 99.0 132.0 109.5 159.0 57.5 60.5 Pour Point (° C.) 53.6 35.0 24.0 Potential Total Sediment (wt %) Ash Content (wt %) 10 Vanadium (wppm) 42.8 167.0 16.5 71.8 93.1 0.3 0.2 0.2 0.2 Sodium (wppm) 9.4 16.1 10.8 7.6 1.1 1.0 1.0 1.0 1.0 Aluminum + Silicon (wppm) 27 40 20 1 Copper (wppm) 0.3 0.4 0.3 0.5 0.3 0.3 0.3 0.3 0.3 Calcium (wppm) 4.69 7.64 6.02 2.77 5.74 0.20 0.20 0.20 0.20 Zinc (wppm) 1.24 3.11 0.91 1.02 2.31 0.40 0.40 0.40 Phosphorus (wppm) 1.16 2.53 1.79 1.35 2.45 1.00 1.00 1.00 1.00 Nickel (wppm) 31.7 67.6 16.1 33.3 37.6 0.3 0.3 0.3 0.3 Iron (wppm) 55 31.4 19.1 7.04 20.7 0.40 0.20 0.20 0.21 Micro Carbon Residue (wt %) 17.16 14.25 17.32 15.57 12.3 4.73 0.04 0.27 0.76 Total Acid Number (mg KOH/kg) 0.10 0.76 0.19 0.18 0.32 0.03 0.02 0.01 0.03 Saturates 10.38 15.7 15.05 13.29 28.62 10.05 56.12 11.21 22.09 Aromatics 70.16 50.06 55.13 54.10 47.43 86.45 41.85 88.78 72.08 Resins 10.32 20.88 18.57 22.1 13.09 2.40 0.53 0 1.77 Aspaltenes 9.12 13.3 11.2 10.5 10.9 1.1 0 0 4.1 CII 0.242 0.409 0.357 0.312 0.652 0.125 1.324 0.126 0.354 Heptane Insolubles 6.42 8.78 8.55 2.43 0.29 0.17 TABLE VII Blend Compositions Component Blnd #1 Blnd #2 Blnd #3 Blnd #4 Blnd #5 Blnd #6 Blnd #7 Resid 1 12.02 23.28 Resid 2 12.84 Resid 3 25.50 Resid 4 24.81 23.36 Resid 5 24.59 Decant Oil 30.66 40.32 53.94 36.94 50.23 47.02 13.59 Vacuum Gas Oil 44.53 46.84 37.56 63.12 Light Cycle Oil 34.05 26.42 Heavy Cycle Oil 28.38 Blend Compositions Component Blnd #8 Blnd #9 Blnd #10 Blnd #11 Blnd #12 Blnd #13 Blnd #14 Resid 1 24.71 Resid 2 23.81 Resid 3 26.29 25.50 22.42 Resid 4 25.89 25.51 Resid 5 Decant Oil 42.35 57.12 36.94 16.24 41.76 32.00 13.70 Vacuum Gas Oil 32.95 16.59 37.56 61.33 32.35 42.49 62.49 Light Cycle Oil Heavy Cycle Oil ..

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$Red light inhibits type I collagen degradation via AKT/NRF2/HO-1 signaling pathway activation A, B: qPCR analysis of NRF2 (A) and HO-1 (B) mRNA expression in mouse skin tissue following red light treatment. n =4. C: Representative western blot assays of p-AKT, HO-1, NRF2, and type I collagen expression in aged mice treated with red light, API-2, or both. D: Western blot analysis of nuclear-cytoplasmic separation in skin tissue. Cytosolic and nuclear extracts were immunoblotted for NRF2, while GAPDH and histone H3 proteins were probed to confirm thorough separation of the cytosolic and nuclear fractions. E: Representative western blot analysis of type I collagen expression in skin tissue after red light treatment with or without HO-1i. F: Representative immunofluorescence images of type I collagen in skin sections from aged mice after 28 days of red light treatment with or without HO-1i. Scale bar: 50 μm. G: Heatmaps showing relative expression levels of DEGs involved in various MMPs. n =3. H: qPCR analysis of MMP3, MMP9, MMP12, and MMP13 mRNA expression in mouse skin tissue following red light treatment, with and without HO-1i. *** : P <0.001; **** : P <0.0001 compared to control group, and #### : P <0.0001 compared to indicated groups, one-way ANOVA for multiple groups. Data are mean±SEM.$

Journal: Zoological Research

Article Title: Red light promotes dermis-epidermis remodeling via TGFβ and AKT-mediated collagen dynamics in naturally aging mice

doi: 10.24272/j.issn.2095-8137.2024.405

Figure Lengend Snippet: Red light inhibits type I collagen degradation via AKT/NRF2/HO-1 signaling pathway activation A, B: qPCR analysis of NRF2 (A) and HO-1 (B) mRNA expression in mouse skin tissue following red light treatment. n =4. C: Representative western blot assays of p-AKT, HO-1, NRF2, and type I collagen expression in aged mice treated with red light, API-2, or both. D: Western blot analysis of nuclear-cytoplasmic separation in skin tissue. Cytosolic and nuclear extracts were immunoblotted for NRF2, while GAPDH and histone H3 proteins were probed to confirm thorough separation of the cytosolic and nuclear fractions. E: Representative western blot analysis of type I collagen expression in skin tissue after red light treatment with or without HO-1i. F: Representative immunofluorescence images of type I collagen in skin sections from aged mice after 28 days of red light treatment with or without HO-1i. Scale bar: 50 μm. G: Heatmaps showing relative expression levels of DEGs involved in various MMPs. n =3. H: qPCR analysis of MMP3, MMP9, MMP12, and MMP13 mRNA expression in mouse skin tissue following red light treatment, with and without HO-1i. *** : P <0.001; **** : P <0.0001 compared to control group, and #### : P <0.0001 compared to indicated groups, one-way ANOVA for multiple groups. Data are mean±SEM.

Article Snippet: To inhibit AKT, mice received intraperitoneal (i.p.) injections of API-2 (HY-15457, MCE, USA) at a dose of 2 mg/kg every 48 h. To inhibit TGFβ, mice received i.p. injections of SB431542 (HY-10431, MCE, USA), a TGFβR1 inhibitor, at a dose of 10 mg/kg every 48 h. To inhibit cAMP, mice received i.p. injections of SQ22536 (HY-100396, MCE, USA), an adenylate cyclase (AC) inhibitor, at a dose of 10 mg/kg every 48 h. To inhibit HO-1, mice received i.p. injections of HO-1i (HY-111798A, MCE, USA) at a dose of 1.357 mg/kg every 48 h. To inhibit intracellular free Ca 2+ , mice received i.p. injections of diltiazem (HY-B0632, MCE, USA) at a dose of 20 mg/kg every 48 h. To inhibit reactive oxygen species (ROS), mice received daily topical application of 100 mg/mL ascorbic acid (HY-B0166, MCE, USA), applied to the skin 30 min after red light exposure.

Techniques: Activation Assay, Expressing, Western Blot, Immunofluorescence, Control